Medical Sources

PubMed

Myricetin was identified as a direct chemical inhibitor of a key SARS coronavirus enzyme, showing potential as a natural antiviral agent against the coronavirus family.

PubMed

This study further explored the use of natural compounds including myricetin as inhibitors of the SARS coronavirus, identifying the viral helicase enzyme as a promising drug target.

PubMed

Myricetin protected heart muscle cells from inflammatory injury, suggesting a role in reducing cardiac inflammation.

PubMed

Dihydromyricetin (Myricetin) was shown to suppress joint inflammation in arthritis by blocking a key inflammatory signaling pathway.

PubMed

A broad review of Myricitrin (a form of Myricetin) documenting its wide-ranging anti-inflammatory and antioxidant molecular effects throughout the body.

PubMed

A myricetin-derived compound demonstrated strong effectiveness in reducing chronic colon inflammation, pointing to Myricetin's therapeutic potential for inflammatory bowel conditions.

PubMed

Using advanced gene-expression analysis, this study mapped the specific proteins and pathways through which myricetin delivers its anti-inflammatory effects.

PubMed

Demonstrated significant anti-inflammatory activity in a myricetin-rich plant extract tested in animal models.

PubMed

Myricetin was shown to reduce severe tissue inflammation and repair biological barriers damaged by inflammatory insult.

PubMed

Myricetin suppressed the skin-based inflammatory responses that drive acne, supporting its use in skin health applications.

PubMed

Dihydromyricetin from Moyeam was found to protect blood vessels from the abnormal tissue buildup that contributes to arterial narrowing and heart disease.

PubMed

Dihydromyricetin protected the inner lining of blood vessels from inflammation-induced dysfunction, a key factor in cardiovascular disease.

PubMed

Dihydromyricetin reduced the formation of "foam cells" — the cholesterol-laden cells that build up in arteries and drive atherosclerosis.

PubMed

In an animal model of atherosclerosis, Dihydromyricetin significantly reduced arterial plaque development, demonstrating a direct anti-atherosclerotic effect.

PubMed

Myricetin demonstrated a significant protective effect against chemically-induced breast cancer in animal models, reducing tumor development.

PubMed

Myricetin was shown to stabilize a specific DNA structure (G-quadruplex) that helps suppress cancer cell replication — revealing a novel anticancer mechanism.

PubMed

This structural study showed how myricetin binds to and inhibits a bacterial protein involved in DNA replication, with implications for targeting similar pathways in cancer cells.

PubMed

Myricetin was found to block multiple enzymes (kinases) that drive tumor growth, suggesting broad-spectrum anticancer potential across several cancer types.

PubMed

Myricetin selectively triggered cancer cell death in triple-negative breast cancer — one of the most difficult-to-treat forms — through a targeted oxidative mechanism.

PubMed

Myricetin was shown to reduce the spread (metastasis) of breast cancer cells by blocking the enzymes that allow tumors to invade surrounding tissue.

PubMed

Myricetin inhibited a key enzyme that drives the growth of estrogen receptor-positive (ER+) breast cancer cells, pointing to its potential as a natural chemopreventive agent.

PubMed

Ampelopsin (Myricetin from Moyeam) induced programmed cell death in human lung cancer cells by disrupting the structural machinery cancer cells use to divide.

PubMed

Ampelopsin (Myricetin) triggered cancer cell death in human brain tumor (glioma) cells through multiple simultaneous pathways, showing strong anticancer activity.

PubMed

Myricetin reduced the ability of bile duct cancer cells to migrate and invade surrounding tissue, directly inhibiting cancer metastasis.

PubMed

Myricetin was shown to significantly lower blood pressure and improve metabolic markers in an animal model of diet-induced hypertension.

PubMed

In a second hypertension model, myricetin again demonstrated a meaningful blood pressure-lowering effect, reinforcing its potential as a natural antihypertensive.

PubMed

Dihydromyricetin protected against bone loss by suppressing the cells responsible for bone breakdown — relevant to both osteoporosis and arthritis-related joint deterioration.

PubMed

Dihydromyricetin was shown to block and even reverse the protein clumping (α-synuclein aggregation) that is a hallmark of Parkinson's disease, protecting brain neurons from damage.

PubMed

In a human clinical study, Moyeam supplementation measurably improved blood sugar control in type 2 diabetes patients — a rare and significant human trial result.

PubMed

Beyond blood sugar, Dihydromyricetin was found to protect the brain from the cognitive decline associated with type 2 diabetes, reducing oxidative stress and boosting neuroprotective factors.

PubMed

Dihydromyricetin suppressed the inflammatory cascade responsible for acute pancreatitis, reducing pancreatic tissue damage in an experimental model.

PubMed

A comprehensive pharmacological study found that Moyeam (Ampelopsis grossedentata) exerted significant anti-inflammatory effects on colitis, helping to protect and heal the colon lining.

PubMed

Myricetin reduced the inflammatory responses in skin cells that trigger acne breakouts, supporting its use as a topical and dietary skin treatment.

PubMed

Dihydromyricetin counteracted steroid-induced muscle wasting by restoring mitochondrial health in muscle cells — relevant for athletes and those recovering from illness or medication.

PubMed

Dihydromyricetin significantly reduced lung inflammation and tissue damage in an acute lung injury model, pointing to its potential value in respiratory conditions.

PubMed

This study on a closely related flavonoid (gossypetin) demonstrated protective cardiovascular and cellular regenerative effects, supporting the broader flavonol family's health role.

PubMed

Gossypetin (structurally related to myricetin) was shown to prevent LDL cholesterol oxidation and the foam cell formation that causes arterial plaque.

PubMed

Dihydromyricetin protected brain function and reduced inflammation caused by lead toxicity, demonstrating a neuroprotective effect against heavy metal poisoning.

PubMed

Tea polyphenols including kaempferol demonstrated direct antiviral activity against both Influenza A and B viruses, with kaempferol among the most effective compounds tested.

PubMed

Kaempferol inhibited the growth of ER+ breast cancer cells by cutting off their glucose supply — effectively starving the tumor cells of the energy they need to proliferate.

PubMed

Kaempferol blocked kidney cancer cells from migrating and invading surrounding tissue by suppressing two key cancer-promoting signaling pathways.

PubMed

Kaempferol was found to boost levels of Coenzyme Q (an essential cellular energy molecule) in kidney cells, supporting kidney function and energy metabolism.

PubMed

Kaempferol from Eagle Tea reduced kidney damage caused by diabetes by blocking a specific inflammatory signaling pathway, supporting kidney health in diabetic patients.

PubMed

Hawk Tea (Eagle Tea, Litsea coreana) demonstrated strong antioxidant activity and the ability to reduce blood sugar spikes after meals, supporting metabolic health.

PubMed

A comprehensive scientific review documenting the full range of medicinal compounds and proven health effects of Eagle Tea's source plant, Litsea coreana.

PubMed

Eagle Tea's flavonoids demonstrated strong antioxidant effects and significantly reduced brain damage from stroke-like ischemic injury in animal models.

PubMed

Eagle Tea's flavonoids reduced a key inflammatory protein (TNF-α) in arthritis models, demonstrating a meaningful anti-inflammatory effect on joint disease.

PubMed

Newly isolated compounds from Eagle Tea leaves showed significant anti-inflammatory activity, adding to the growing body of evidence for its therapeutic value.

PubMed

Eagle Tea (Hawk Tea) was shown to meaningfully protect the liver from toxic chemical-induced damage, supporting its role in liver health.

PubMed

Eagle Tea's flavonoids protected against the development of alcoholic fatty liver disease, reducing fat accumulation in liver tissue.

PubMed

Eagle Tea's total flavonoid content demonstrated significant antidiabetic effects in a type 2 diabetes animal model, improving insulin sensitivity and glucose metabolism.

PubMed

Eagle Tea showed protective effects against liver fibrosis — the scarring process that can progress to cirrhosis — in an animal model of liver disease.

PubMed

Eagle Tea's flavonoids prevented the buildup of fat in the liver caused by a high-fat diet, offering a natural approach to combating non-alcoholic fatty liver disease.

PubMed

Demonstrated that gossypetin (a flavonoid found in hibiscus) protects blood vessel walls from injury through cellular self-cleaning (autophagy) mechanisms, supporting cardiovascular health.

PubMed

Gossypetin found in the Chrysanthemum/Hibiscus blend was shown to prevent LDL ("bad") cholesterol oxidation — the key process that leads to artery-clogging plaque formation.

PubMed

SH003 triggered a form of cancer cell self-destruction (autophagic cell death) in gastric cancer cells even under low-oxygen conditions that typically allow tumors to survive.

PubMed

SH003 halted cervical cancer cell division at a key checkpoint and triggered programmed cell death, demonstrating anti-cancer activity against cervical cancer.

PubMed

SH003 overcame chemotherapy resistance in breast cancer cells by blocking a signaling pathway (STAT3) that tumors use to evade treatment.

PubMed

SH003 made paclitaxel chemotherapy more effective in drug-resistant breast cancer cells by suppressing a protein that normally pumps the drug out of cancer cells.

PubMed

SH003 inhibited breast cancer tumor growth by disrupting the cancer cells' waste-disposal system, causing a buildup of proteins that triggered cell death.

PubMed

SH003 suppressed both primary tumor growth and the spread (metastasis) of an aggressive form of breast cancer by blocking a key inflammatory cancer-promoting signaling axis.

PubMed

When combined with the chemotherapy drug doxorubicin, SH003 produced a synergistic effect — meaning the combination was significantly more powerful than either treatment alone against triple-negative breast cancer.

PubMed

SH003 selectively triggered programmed cell death specifically in triple-negative breast cancer cells — the most aggressive and treatment-resistant subtype — while sparing healthy cells.

PubMed

SH003 cut off the blood supply to tumors by preventing the growth factor (VEGF) that tumors use to create new blood vessels from binding to its receptor — effectively starving the tumor.